Preliminary studies point out conolidine might inhibit distinct ion channels, decreasing neuronal excitability and restricting soreness indicators. This mechanism is particularly relevant in neuropathic soreness, where abnormal signaling triggers persistent discomfort. Moreover, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Comp... https://mitchk036khe6.wikinarration.com/user